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erature reading lesson to those who
have made these claims. See Proceedings of the Royal Society of
London, Series B, Volume 155, pages 26 to 54 (1961). Also see US
You will note while reading these articles detailing
how to get lysergic amide production in a culture medium that these
guys had to scour the globe to find that rare strain of claviceps fungus
that will cooperate in this manner. The vast majority of claviceps
fungi just will not produce these alkaloids while being cultured. See
the following articles to convince yourself of just how futile it is to
collect a wild strain of claviceps and try to get it to produce lysergic
acid amides in culture: Ann. Rep. Takeda Res. Lab Volume 10, page 73
(1951); and Farmco, Volume 1, page 1 (1946); also Arch. Pharm. Berl.
Volume 273, page 348 (1935); also American Journal of
Practical LSD Manufacture
Botany, Volume 18, page 50 (1931); also Journal of the American
Pharmacy Association Volume 40, page 434 (1951); also US patent
2,809,920; also Canadian Journal of Microbiology, Volume 3, page
55 (1957), and Volume 4, page 611 (1958) and Volume 6, page 355
(1960); also Journal of the American Pharmacy Society Volume 44,
page 736 (1955).
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this matter disposed of, it is time to move on to what
actually are viable sources of lysergic acid amides for the production of
LSD. This is the farming end of the acid business. It is only through
raising ergot-infested rye, or growing morning glories and Hawaiian
baby woodrose that the required feedstocks of lysergic compounds
can be obtained without making a target of oneself.
I have for years
seen ads in High Times offering morning glory seeds and Hawaiian
baby woodrose seeds for sale, but these are offered in small amounts at
high prices. I would bet my bottom dollar that these outfits, if they are
not front operations, will at least report to the heat any large orders they
get. To avoid detection, the aspiring LSD manufacturer must be ready
to get his hands dirty, and spend some time as a farmer.
The most difficult farming choice, and as luck would have it, the
one that gives the purest acid, is to grow a patch of ergot-infested rye.
The reason why ergot is superior to growing morning glory seeds or
woodrose seeds is that these seeds have a considerable amount of
another type of alkaloid in them besides the ones that yield lysergic
acid. These other alkaloids are of the clavine type, meaning that they
have the lysergic-acid skeleton, but lack the carboxyl grouping. In its
place will be a methyl grouping, an alcohol grouping, a methyl
alcohol grouping or combinations of the above. These clavine
alkaloids will likely be carried all the way through into the product,
producing both the GIGO situation during the synthetic operations
and a contaminated product when finished. I will present my ideas on
how to remove them, but they are best avoided in the first place.
Ergot is the name given to a dark brow
In America too, over 100,000 people died last year from prescription drugs, (including 1,000 suiciding on Prozac alone!) and not one death from herbal medicine!
Yet, here in Australia, Coltsfoot is still prohibited
We keep all our online customers up to date, with the monthly Xtreme newsletter, featuring new products and developments which might interest you nd tremors, sometimes resulting in death. The
syndrome affects sheep that have eaten high alkaloid phalaris. On
autopsy, after naturally and experimentally induced phalaris
staggers in sheep, portions of the lower brain are seen to be
damaged and, oddly enough, tinted blue. The responsible agents
are the alkaloids contained in the various phalaris species.
There are whispered rumors that phalaris also contain beta
carbolines, a type of MAOI. If so,it may be that sheep, in eating
large quantities of phalaris, also obtain a dose of MAOI, making
the already large dose (pounds of phalaris could easily be eaten
by a sheep or cow in a day), of DMT active. However, MAOIs are
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not required for sheep death. Studies show that controlled
injections of pure DMT, at human recreational doses, kill sheep.
Why sheep and humans respond differently to DMT is still an open
question here. It may be that it is not an important question,
but the possibility of human death on phalaris still looms.
XXXXXXXXXX DOSES XXXXXXXXXX
There seems to be a variety of dosages (of pure drug)
suggested in the literature. Table 2 lists some of the reported
dosage levels. It should be noted that levels that are listed in
table two are within the range reported to cause "phalaris
staggers" in sheep and cause them to die.
Table 2 Jonathan Net Peter Bufotenine
REPORTED DOSES Ott Lore Gessner Literature
DMT 1mg/kg 60mg --- ---
5-Me-DMT .25mg/kg 5mg 5-10 mg ---
5-OH-DMT NONE! --- --- 16mg
NOTE: These 'reported doses' are non-lethal dosages given to
humans in the literature as referenced. THIS AUTHOR MAKES NO
RECOMMENDATIONS! The "mg/kg" means one mg of drug for every kg of
Using Ott's levels, approximately .5g of Phalaris Tuberosa
per kilogram body weight should yield one dose of 50% DMT and 50%
5-Me-DMT. Using the net lore, 25g of Phalaris Tuberosa would
yield 1 dose of 5-Me-DMT with less than 1/2 dose of DMT: still a
fairly strong dose when totaled. These doses are based on plants
that have not had their alkaloid levels boosted.
Remember, there is WIDE variation of alkaloid levels. One
week a plant may yield a minimal dose, the next week it may blow
your mind away.
XXXXXXXXXX BOOSTING ALKALOIDS XXXXXXXXXXX
The scientific literature, intended to solve the problem of
"Phalaris staggers" in sheep has revealed the growth parameters
that will optimize the alkaloid content of Phalaris Tuberosa
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